If we were in charge, however, we on medical grounds would not use vaccination (or other herd immunity) programs. This is because a vaccine exerts selective pressure on the virus (similar to how predators exert selective pressure on prey) thus leading to virus mutating into more complex forms that enable it to survive the antibodies generated by the vaccine in the long-term, which will later necessitate a new vaccine (similar to how prey exert selective pressure on predators) that generates more complex antibodies, which then does the same thing to the new virus in an endless cycle. This is the typical Western approach to all problems, which we ideologically reject.
The correct way to counter a virus (used since ancient times) is by strict lockdown to prevent the infected from infecting anyone else. Deprived of a supply of new hosts, the virus is automatically doomed, and we just have to wait it out. It is that simple.
The problem is, we aren't the only prey. For the virus to be genuinely "doomed", all its host species would need to perform strict lockdown (eradicating animal farming will definitely help). We'll always be in an arms race against zoonotic viruses until then. But I agree, vaccination only levels up the game. Strict lockdown and hygiene is a safer way of controlling viruses, it too generates selection pressure, but much slower since it's effectively neutering the virus so they have little chance to mutate. But unless we maintain a baseline biological immunity, every virus will be extremely contagious and destructive, undermining our methods. Ideal immunity would replicate lockdown/hygiene on a biological level, so rather than being "adaptive" and developing specific antibodies, it prevents the virus getting a foothold in the first place.
Have you looked into the child vs adult immune system? They function fundamentally differently and in many cases childrens' are more effective. Children have a weaker adaptive immune response and a stronger innate immune response.
Compared with adults, children recently infected with SARS-CoV-2 have also been found to have higher levels of activated neutrophils, cells that are on the front line in the response to unfamiliar invaders. Neutrophils ingest viral particles before they have a chance to replicate, says Melanie Neeland, an immunologist at the Murdoch Children’s Research Institute (MCRI) in Melbourne, who led the work. Furthermore, they become less effective with age.